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dylight 488 antibody kit  (Vector Laboratories)


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    Structured Review

    Vector Laboratories dylight 488 antibody kit
    Dylight 488 Antibody Kit, supplied by Vector Laboratories, used in various techniques. Bioz Stars score: 94/100, based on 29 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/DK-1488/VectaFluor+Excel+Amplified+DyLight+488+Anti-Rabbit+IgG+Kit/pmc13102498-133-36-41
    Average 94 stars, based on 29 article reviews
    dylight 488 antibody kit - by Bioz Stars, 2026-10
    94/100 stars

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    Related Articles

    Amplification:

    Article Title: Intradiscal Cutibacterium acnes Sustains Modic Type 1‐Like Lesions Over Time in a Rat Lumbar Endplate Injury Model
    Article Snippet: Normal mouse serum (#NC494H, Biocare Medical LLC, Pacheco, CA, USA) or normal rabbit serum, respectively, was used as a negative control. .. A secondary antibody for SubP was carried out with a VectaFluor Excel Amplified anti‐mouse IgG, Dylight 488 antibody kit (#DK‐2488, Vector Laboratories Inc., Burlingame, CA, USA), and for GFAP with a VectaFluor Excel Amplified anti‐rabbit IgG, Dylight 488 antibody kit (#DK‐1488, Vector Laboratories Inc., Burlingame, CA, USA), according to manufacturer's instructions. .. Sections were then stained with NeuroTrace 530/615 red fluorescent Nissl stain (1:200 dilution, # N21482 , ThermoFisher Scientific, Waltham, MA, USA) to visualize the neurons and glial cells.

    Article Title: AAV capsids and compositions containing same
    Article Snippet: .. The signal for the rabbit antibody against CD31 was enhanced using a VectaFluorTM Excel Amplified DyLight® 488 Anti-Rabbit IgG kit according to the manufacturer's protocol (Vector Labs). .. Fluorescence and brightfield microscopy images were taken with a Nikon Eclipse TiE microscope.

    Article Title: 2-Chloroethanol induces hepatic toxicity by disrupting endoplasmic reticulum homeostasis ameliorated by dimethyl sulfoxide.
    Article Snippet: 2-Chloroethanol (2CE), a metabolite of ethylene oxide, vinyl chloride, and 1,2-dichloroethene, induces acute liver injury, but its mechanisms remain poorly defined.. In this study, we demonstrate that 2CE disrupts endoplasmic reticulum (ER) homeostasis and activates unfolded protein response (UPR) signaling in both mouse liver and H4IIEC3 hepatoma cells.. While in vivo eIF2α–CHOP signaling predominated, all three canonical UPR branches—IRE1α–XBP1s, PERK–eIF2α–CHOP, and ATF6—were activated in vitro.

    Article Title: AAV capsids and compositions containing same
    Article Snippet: .. The signal for the rabbit antibody against CD31 was enhanced using a VectaFluorTM Excel Amplified DyLight® 488 Anti-Rabbit IgG kit according to the manufacturer's protocol (Vector Labs). .. Fluorescence and brightfield microscopy images were taken with a Nikon Eclipse TiE microscope.

    Article Title: A new paradigm of islet adaptations in human pregnancy: insights from immunohistochemistry and proteomics
    Article Snippet: .. For the anti-PRLR antibody the VectaFluorTM Excel Amplified Anti-Rabbit IgG, DyLightTM 488 Antibody Kit (Vector Laboratories, cat. no. DK-1488) was used, as per manufacturer instructions, in lieu of 5% goat serum and the secondary antibodies to amplify the PRLR signal. .. DAPI (1 μg/mL; Thermo Fischer Scientific, cat. no. 62248) was used to label nuclei.

    Staining:

    Article Title: 2-Chloroethanol induces hepatic toxicity by disrupting endoplasmic reticulum homeostasis ameliorated by dimethyl sulfoxide.
    Article Snippet: 2-Chloroethanol (2CE), a metabolite of ethylene oxide, vinyl chloride, and 1,2-dichloroethene, induces acute liver injury, but its mechanisms remain poorly defined.. In this study, we demonstrate that 2CE disrupts endoplasmic reticulum (ER) homeostasis and activates unfolded protein response (UPR) signaling in both mouse liver and H4IIEC3 hepatoma cells.. While in vivo eIF2α–CHOP signaling predominated, all three canonical UPR branches—IRE1α–XBP1s, PERK–eIF2α–CHOP, and ATF6—were activated in vitro.

    Incubation:

    Article Title: 2-Chloroethanol induces hepatic toxicity by disrupting endoplasmic reticulum homeostasis ameliorated by dimethyl sulfoxide.
    Article Snippet: 2-Chloroethanol (2CE), a metabolite of ethylene oxide, vinyl chloride, and 1,2-dichloroethene, induces acute liver injury, but its mechanisms remain poorly defined.. In this study, we demonstrate that 2CE disrupts endoplasmic reticulum (ER) homeostasis and activates unfolded protein response (UPR) signaling in both mouse liver and H4IIEC3 hepatoma cells.. While in vivo eIF2α–CHOP signaling predominated, all three canonical UPR branches—IRE1α–XBP1s, PERK–eIF2α–CHOP, and ATF6—were activated in vitro.



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